This is a great line, thanks! Going to share this with my distributed database reading group
During that time though, nothing I read and nobody explained to me what keeps the heart beating despite all manner of possible failures. 10/10 was definitely worth the read.
The symptoms of a heart attack and a panic attack are annoyingly similar. Whereas people qualified to diagnose and treat one are generally nowhere near the people qualified for the other.
The wonderfully comical thing about mine is they’re stress related. So the first one happened, made me a little stressed because I thought something was wrong, which caused more of them, which made me even more worried, etc.
I'm not into any competitions or races. I run for health purposes only, so I'm okay losing some fitness. If the symptoms disappear, I'll certainly ramp up again. If they don't, I'll play it by ear :)
My understanding is that 35-50% of endurance athletes have what I have (including symptoms), and they continue to train without any harmful effects.
In my case, the PVC disappears during exercise and comes back when I take a rest after exercising. I just wonder if you had any comparisons between rest vs. elevated heart rate scenarios. Now I'm super curious how elevated heart rate might change the clock rates for those nodes.
From what I've read, when we're exercising (or sick or stressed) the heart rate gets overridden by the SNS (part of the autonomic nervous system). The SA node can only maintain resting heart rate, and once there's extra demands from the body, the SNS takes over (controlled by the brain, but involuntary).
It seems to be a good sign that PVCs disappear during exercise based on the studies I found.
Only after a couple of weeks of running about 20km, did I start seeing my resting heart rate drop to the high 40s. After that, it gradually lowered over the next 18 months or so.
Furthermore, an article about a study back in 2008 [1]:
> At the end of the 90-day study period, both groups had significant overall increases in the size of their hearts. For endurance athletes, the left and right ventricles — the chambers that send blood into the aorta and to the lungs, respectively — expanded. In contrast, the heart muscle of the strength athletes tended to thicken, a phenomenon that appeared to be confined to the left ventricle. The most significant functional differences related to the relaxation of the heart muscle between beats — which increased in the endurance athletes but decreased in strength athletes, while still remaining within normal ranges.
> “We were quite surprised by both the magnitude of changes over a relatively short period and by how great the differences were between the two groups of athletes,” Baggish says. “The functional differences raise questions about the potential impact of long-term training, which should be followed up in future studies.”
[1] https://news.harvard.edu/gazette/story/2008/04/exercise-chan...