While the analyses above suggest that SARS-CoV-2 may bind human ACE2 with high affinity, computational analyses predict that the interaction is not ideal7 and that the RBD sequence is different from those shown in SARS-CoV to be optimal for receptor binding7,11. Thus, the high-affinity binding of the SARS-CoV-2 spike protein to human ACE2 is most likely the result of natural selection on a human or human-like ACE2 that permits another optimal binding solution to arise. This is strong evidence that SARS-CoV-2 is not the product of purposeful manipulation.
But again, the poster clarified this only addresses the engineering of the virus, not the the other theories that center around lax precautions at the lab.
So, these threads do devolve into a predictable pattern, but I’m compelled to bring up instances where viruses escaped from Chinese labs before:
https://www.the-scientist.com/news-analysis/sars-escaped-bei...
I believe there were a few more instances too (not mentioned in the article).