The reason this isn't getting funded is because it's extremely unlikely to work in reality. They've engineered synthetic protein sensors of dsRNA that induce apoptosis on detection - this mimics the natural viral detection systems that all cells use to prevent viral infection. The argued benefit is that this artificial protein avoids the common hacks that many viruses use to circumvent the natural detection pathways. For this to work you have to (1) inject a ton of this foreign protein into the blood, (2) have it persist and be taken up by many cells in an organism, (3) not elicit a negative immune reaction against cells expressing this foreign beast on their MHCs (4 - for prophylaxis) have it persist long enough to provide realistic protection against future viral infection events (5) hope that the distribution of protein-transduced cells overlaps enough with the natural viral targets to provide some clinical utility.

The costs of trying to turn a novel therapeutic approach into a real therapy are extremely high - hundreds of millions of dollars. I'm unaware of any approved therapy that utilizes protein transduction of cells - I also suspect existing protein transduction methods aren't very efficient. There is a tiny pile of evidence that this method "works" in vitro in cultured cell models of infection, I can imagine a hundred ways it will fail in bodies.

There's a reason some ideas are left unexplored by industry.