It's clear that nicotine binds to those receptors, but exactly how that plays out is far more complex in practice, depending on a whole host of variables.

- Presence or absence of other nicotinic agonists, inverse agonists, and antagonists, and relative concentrations of each

- Effects vary wildly with nicotine concentration levels in plasma/interstitial fluid, as if it's an entirely different drug depending on how much is present

- etc.

You're drawing a direct line from one agonist the paper only mentions once to a psychological effect that the paper doesn't even describe. You may or may not be right, but this paper is at best a citation for a separate study to test your hypothesis.