I don't think this works on astigmatism though.
There are dopaminergic amacrine cells within the retina and dopamine receptor much more widely.
Here is a reference with some of original citations:
> Dopamine is a key neurotransmitter in the signaling cascade controlling ocular refractive development, but the exact role and site of action of dopamine D1 receptors (D1Rs) involved in myopia remains unclear.
[.. snip ..]
> Therefore, activation of D1Rs, specifically retinal D1Rs, inhibits myopia development in mice. These results also suggest that multiple dopamine D1R mechanisms play roles in emmetropization and myopia development.
1) Your prescription.
2) What your vision is like with the best correction.
It also is possible to have your vision improve without treatment. I have some astigmatism, it's simply impossible to fully correct. My prescription changes a lot, it's possible for the astigmatism to be a bit less of an issue on a particular exam to the point that the best line I can read sometimes changes.