I have to argue against this - humbly as an expert with several degrees in microbiology, immunology, genetics, host-pathogen interaction, university lecturing, etc. The genetic basis of immunology is amazingly complex. To assign 90% autoinflammatory type disease triggered by pathogens is an unfounded assertion. There are many strong examples of monogenic causes of complex phenotypes, while separately the clinical microbiology explanation of many diseases is also perfectly clear. I am certain that there will be many pathogen triggers, as you say, but the number 90% does not make sense based on the last 100 years of the science.