Its know that most viruses trend towards increased transmissibility and decreased lethality.
Killing your host is an awful strategy for continued existence if you don't have your own lipid bilayer.
Killing your host is an awful strategy for continued existence if you don't have your own lipid bilayer.
The virus does not decide a strategy, it is random mutation and selection and even if it is less likely it might be that we could have a variant that is super transmissible and super deadly. This effect could be obtain in the delay between transmission and visible effects on human body. Example: virus is transmissible with N days before we can see signs of infection.
Of course we can fight back by testing everyone weekly even if they show no signs.
Help me understand how a virus can replicate without being detected but can also increase its lethality.
We already have an asymptomatic and infectious incubation period. If that kept stretching longer then you are delaying the lytic cycle even longer? Or does propogation and ultimately viral load also happen as a slower burn? Eventually you will have damaged too many cells.
I am not up to date with latest research but it seemed to be that SARS-CoV-2 is a virus that can be transmitted before symptoms onset.
Regarding replication without being detected but also increased lethality this you might be right that doing the math maybe the hypothesis I present seems far fetched.
Still I guess that a slight bump in mortality will create a lot of deaths due to its transmission.
Of course like I said I am just expressing an opinion that might be very wrong.
No virus has ever done this.