The guy quoted for said statement is a professor of medicine of a reputable university, so he does have the authority thing sorted out.
Also, I feel the quote means something different than the original interpretation. After reading the article, it seems that John Bell simply states that a) long Covid is "more complicated than people assume", and b) it's incidence is lower than initial (and conservative) estimates.
The article mentions that "half of people suffering from long Covid may not have it."
And that's pretty much it.
There is no harm in improving our understanding of the real world effects of a disease.
That’s fallacious. There are loads of professors spewing bullshit, including about their field, including at Oxford. It says nothing about the validity of the point they are making. Particularly after the point has been filtered through journalists with a questionable understanding of science.
No, not really. The fallacious bit is blindly and outright dismissing any statement, along with attacking and accusing anything a researcher says as being "spewing bullshit", just because you didn't liked hearing it.
Eve though I agree that appeals to authority are no way to get close to the truth, what we are seeing here is pure denialism and a primal anti-intellectual attitude directed towards a researcher from a reputable university just because he dared point out that initial conservative estimates might have been conservative.
To be quite clear, John Bell does not dictate the truth just because he is a professor of medicine at Oxford. But that is not the problem. The problem is actually trying to attack him just because he had the audacity to quote statistics on the real-world incidence of long Covid and state that initial estimates were off.
Sounds like the BMJ dont trust this chap and are questioning his financial interests. Therefore I would question his motives with regard to this article, he isnt very forthcoming which is usually indicative of having something to hide.
The article you mentioned only refers to stock portfolio and potential conflicts of interest regarding policy decisions on performing antibody tests.
The statement that was quoted in this discussion is about an entirely unrelated subject: the prevalence of long Covid. The initial claim is that the statement is supported by data from the Office for National Statistics.
If you want to question John Bell's claim regarding the prevalence of long Covid, just check if his statement is corroborated by the data. Either it's supported or not.
There is a very good way to verify the claims: check the data.
Dragging in questions regarding antibody test policies, stock portfolios, and potential conflicts of interest just sound like cheap character assassination attempts that have absolutely no relation with the prevalence of long Covid.
Don't accept things blindly because you agree with them (or the converse), "be a good judge of who is a good judge".
If that's what you're going with then you only need to verify the data to either refute or verify his claim.
Everything else is just noise at this point.
You didn't even bothered to look, did you?
https://www.ons.gov.uk/peoplepopulationandcommunity/healthan...
That is not nearly good enough.
This is HN. We should be posting peer-reviewed research results, not just-any-professor-in-the-field's-feelings.
That's a completely baseless statement, and there is some serious fuckery going on here, either with the article, or the supposed expert they're citing.
Remember these are the same media organisations that will deny climate change and find scientists who will push that agenda for them. Just because they have a pulpit, it doesn't make them correct.
Low energy
Headaches
Upset stomach, including diarrhea, constipation, and nausea
Aches, pains, and tense muscles
Chest pain and rapid heartbeat
Insomnia
Frequent colds and infections
Loss of sexual desire and/or ability
Nervousness and shaking, ringing in the ear, cold or sweaty hands and feet
Dry mouth and difficulty swallowing
Clenched jaw and grinding teeth
Sounds like all of the physical symptoms of so-called long-COVID.
On top of the stress that would undoubtedly afflict a significant fraction of COVID patients, due to the massively inflated fears surrounding COVID, is the effects of physical isolation.
One of the requirements for all COVID positive cases is two weeks of total physical isolation. Numerous studies have shown that humans being physically isolated has significant negative health effects. No in person contact. No physical touch. For two weeks. All COVID positive cases, even asymptomatic ones.
There are no randomized studies, that control for factors, like the aforementioned psychological distress emanating from fear of COVID, and physical isolation, that suggests COVID causes these long-term symptoms. There are a host of potential confounding factors that could explain these correlations better than damage from the COVID infection.
Notice how extreme rigor is demanded for the ivermectin studies but then really just mere correlations is enough to draw scare mongering conclusions about COVID.
But: pretty much every single anecdote I've heard (online, or meatspace) tells a very consistent story. Maybe it should be studied. But I guess if some professor at a "prestigious university" says it's all psychosomatic, then I guess not.
I feel you inadvertently tried to misrepresent the symptoms of long Covid.
Please check wikipedia's article on long Covid for an objective and we'll sourced reference on the subject:
https://en.wikipedia.org/wiki/Long_COVID
Among the typical symptoms we have "fatigue, headaches, shortness of breath, anosmia (loss of smell), parosmia (distorted smell), muscle weakness, low fever and cognitive dysfunction".
Feeling fatigued alone does not pass off as long Covid, jus like coughing alone does not pass off as Covid.
For the sake of having a non-inflammatory discussion, please do your best to substantiate and source these sort of claim so that we avoid spreading disinformation.
That is not true. Here is a typical study purportedly showing evidence for long COVID:
https://www.theguardian.com/world/2021/sep/28/covid-37-of-pe...
>>One in three people infected with coronavirus will experience at least one symptom of long Covid, a new study suggests.
The most common symptom associated with "long-COVID" is anxiety..
You've said "study" but you're quoting an article from The Guardian.
Also, it seems you even failed to read your source. Your article does not refer to the incidence of long COVID-19. Your source only refers to a statistical study of the prevalence of any of the symptoms that are attributed to long COVID-19, whose goal is clearly to gather data to help understand the problem, or even if one really exists.
Also, I feel you are either misquoting or being disingenuous. The article you quoted quite clearly states that "The most common symptoms were breathing problems, abdominal symptoms, fatigue, pain and anxiety or depression."
So, a charitable interpretation of your misunderstanding of what you've quoted is that you picked up a reference to a study whose purpose is to gather initial observations on the actual incidence of long Covid, looked at the raw data and unwittingly misinterpreted correlations for consequences.
An article which describes the results of a study..
>>Also, I feel you are either misquoting or being disingenuous. The article you quoted quite clearly states that "The most common symptoms were breathing problems, abdominal symptoms, fatigue, pain and anxiety or depression."
I'm referring to the graph in that article, which shows that anxiety/depression is the most commonly cited symptom, at 15% of survey respondents:
https://i.ibb.co/27yyhGG/long-covid-symptoms.png
>>Your article does not refer to the incidence of long COVID-19.
Yes it does. Long-COVID typically refers to any expression of any of the symptoms listed, following a COVID infection.
Presenting your own editorialized conclusions is not the same as providing a primary source.
The proof is that you've tried to pass off a study on the quantification of the real world correlation of symptoms tentatively linked to long covid as, somehow, proof of a causal relationship, even though nothing of the sort was suggested.
You're inadvertently demonstrated the need to check primary sources, and the role that tabloids play in disseminating misinformation
This is a discussion forum. You seem to have an axe to grind with me that's motivated by more than just what I wrote. Are you this non-charitable and critical of those who are sounding the alarm about COVID? This double standard, motivated by political preconceptions, is how dissenting viewpoints are suppressed, and only one viewpoint - that of COVID hysteria - is allowed to express itself.
>>The proof is that you've tried to pass off a study on the quantification of the real world correlation of symptoms tentatively linked to long covid as, somehow, proof of a causal relationship, even though nothing of the sort was suggested.
To clarify, my criticism is of the many who are in fact taking these preliminary results, and drawing the conclusion that long-COVID is afflicting one third of all COVID patients.
I would also add that while I was certainly a bit depressed by the lockdown, I live in the UK where we are no longer locked down and my mental health has improved greatly. But I am still unable to participate in vigorous exercise, and tire much much more easily than my friends. And this is despite going for regular walks during lockdown.
Finally, I would comment that post viral fatigue is a well established phenomenon for other viral diseases including flu. And has also been noted to be particularly bad in other coronaviruses like the original SARS. So it’s hardly a stretch to think that cocos might have similar effects.
One study on pro-athletes found that 0.6% of cases were confirmed, after cardiac MRI, to have inflammatory heart disease post COVID infection:
https://www.cidrap.umn.edu/news-perspective/2021/03/few-pro-...
This is only 1/10th as much as the percentage who report chest pain. Some caveats: these are young adults, and athletes, so probably in the lowest risk category. Other demographics could face much higher risks.
So I guess the chest pain could be a real symptom of long-COVID, but I am much more skeptical of the other symptoms (e.g. anxiety, depression, etc).
It's also worth noting that heart inflammation from infections like flu will generally subside over time.
>>One could easily make the case that the drastic rise in violent incidents in public spaces across the world has not been due to the effects of local and sporadic lockdowns on psychological health, but more likely due to the partial frontal lobotomies being performed by the virus.
You don't think it could be a result of massive numbers of careers being destroyed, businesses going bankrupt, and people being physically isolated, as governments restrict people's economic and civil liberties?
And isn't it possible that fearful speculation of the type you're engaging in, is creating political support for the restrictions that are creating all of these adverse social effects?
It's also pretty widely known that virtually all viruses can have a post-viral syndrome so if it would be MORE surprising if there wasn't a long covid.
I've had long covid for the last 10 months, and finally seem to be coming out of it. At its worst, I would get "episodes" where my entire body would start buzzing, so extreme that it felt like I was plugged into an electric socket. I can't convey to you how extreme this was, and if anyone were to suggest that this was caused by pandemic stress, they would be liable to a stressed response from my fist.
You use antibody tests to find undiagnosed prior cases, where the patient was unaware they had it during their infection. Then you can compare those who had COVID, without the associated psychosocial impact and two week isolation, to those who did not, to try to determine what long-term impact COVID has.
Also: what if the stress symptoms they're feeling, are due to the body fighting long-covid?