Increased risk of traffic accidents in subjects with latent toxoplasmosis (2002)
ncbi.nlm.nih.gov
ncbi.nlm.nih.gov
Not an expert so don't know how well we understand the mechanism of action, which is crucial.
As far as mechanism of action, T. gondii infects both neurons in the brain and glial cells.
More info here if you want it https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1118145/
It sounds a lot like the explanation for chronic Lyme disease (except with nerve tissue and not fat cells), which is apparently still quite controversial despite having lots of anecdotal evidence to support it.
Also if the parasite is hiding in your fat cells, does that also mean that you can't detect it in a blood test?
https://www.sciencedirect.com/science/article/abs/pii/S00144...
"A total of 252 women with toxoplasmosis, along with 748 controls from Naples, Lausanne, Copenhagen, Oslo, Brussels, and Milan, were interviewed by telephone or in person. Overall, eating raw or undercooked beef, lamb, or other meats; contact with soil; and travel outside the country were major sources of infection."
Cats don't even make the list of major sources of infection. Further,
"The association of cats and human toxoplasmosis is difficult to assess by epidemiological surveys because soil, not the cats, is the main culprit. Oocysts are not found on cat fur and are often buried in soil along with cat faeces.11 Therefore, direct contact with cats is irrelevant with respect to T gondii transmission, and soil contact is universal and difficult to avoid."
On one hand you remind people just how un-impaired various BACs are and that pisses them off.
On the other hand you make mildly sick people look like drunks and it becomes a potential pretext for denying people freedom of movement.
Of course most of driving is so easy that I’m sure a huge percent of the time drunk driving has no consequences, but haven’t we all been in an occasional situation that requires fast reaction to narrowly avoid a crash?