You haven't really given me any math to dismiss. But simply put, I believe the risks of locking down until a vaccine outpace the risks of allowing SARS-CoV-2 to spread normally
in the general population (not in high-risk categories).
Your position also implicitly relies on the notion that SARS-2 isn't spreading right now, but it is, so by the time we get a vaccine we will have a lot of exposure. Data from NY and Sweden implies somewhere around 20% seroprevalence is when we hit herd immunity.
Since you haven't given me much to work on, and I don't feel like digging into the vaccine side, let me give you some research on long-term damage:
["Follow-up Chest CT findings from discharged patients with severe COVID-19: an 83-day observational study"](https://www.researchsquare.com/article/rs-27359/v1) - First Submitted May 4, Published online May 12
> Radiological abnormalities in patients of severe COVID-19 could be completely absorbed with no residual lung injury in more than two months’ follow-up.
https://onlinelibrary.wiley.com/doi/full/10.1046/j.1440-1843... (SARS-1 pathology)
> Preliminary evidence suggests that these lung function abnormalities will improve over time
and here's just one effect of lockdown: loneliness.
https://www.pnas.org/content/pnas/110/15/5797.full.pdf
> mortality was higher among more socially isolated and more lonely participants. However, after adjusting statistically for demographic factors and baseline health, social isolation remained significantly associated with mortality (hazard ratio 1.26, 95% confidence interval, 1.08–1.48 for the top quintile of isolation), but loneliness did not (haz-ard ratio 0.92, 95% confidence interval, 0.78–1.09). The association of social isolation with mortality was unchanged when loneliness was included in the model.