I don't think it's a counterpoint to the study, but rather to the ways the data will drive policy changes.
It's taboo to take the obesity problem seriously. https://en.wikipedia.org/wiki/Fat_acceptance_movement
I don't think it's a counterpoint to the study, but rather to the ways the data will drive policy changes.
It's taboo to take the obesity problem seriously. https://en.wikipedia.org/wiki/Fat_acceptance_movement
It is phenomenal to see such a movement sprout up, as if a chunk of society decided to just give up. I couldn't imagine a serious, equivalent movement on the same scale for alcoholism and cigarette smoking, to cite a few examples. Yet the abuse of food is going to (or is already) cost us far more.
Endocrine system diseases need to be researched more and the general public needs more education on it. Sugar doesn't cause Type-I or Type-II diabetes as it seems many on the Internet believe. And being fat isn't the only way to develop Type II diabetes.
And as my mother says, "I'm not a diabetic, I'm a person who has a disease called diabetes."
I remember as a kid our family doctor straight up telling my parents that they were too fat and they needed to exercise and lose weight. I assume doctors still do this, as it's good health advice, but with "fat shaming" being a thing now, I'm sure that some are more hesitant.
60-80% of diabetes type II cases among westerners are assumed to be a consequence of obesity and related medical conditions.
It is correct that you don't have to be obese to get type-II, but being obese makes it much more likely.
Pre-diabetes can be reversed but once you have diabetes, it's permanent - until we figure out how to regenerate beta cells in the pancreas.
Can someone who has had their diabetes "reversed" via fasting then get normal glucose responses after eating sugar?
The N=1 study you cited states that the subject still needs to take Metformin. i.e., he still has diabetes.
http://care.diabetesjournals.org/content/34/Supplement_2/S36...
>It has been over 10 years since the resolution of type 2 diabetes was observed as an additional outcome of surgical treatment of morbid obesity. Moreover, it has been shown unequivocally that diabetes-related morbidity and mortality have declined significantly postoperatively, and this improvement in diabetes control is long lasting.