People are lazy, but they also do not want to completely ruin their life, so they will wait until they realize they cant wait anymore, doing the minimum amount at the last minute to meet the goal.
People are lazy, but they also do not want to completely ruin their life, so they will wait until they realize they cant wait anymore, doing the minimum amount at the last minute to meet the goal.
To be more clear as to the diagnosis: there is a definite difference between people who don't want to pay attention to certain tasks (the vast majority) and those who cant (the minority that do have legitimate ADHD).
Also having the capacity to concentrate but refraining from using it is an active choice; people doing that won't be complaining that they can't focus.
A premise for Adhd is that, for some reason, developement of the latter is delayed causing an apparent lack of the former. The inpairment is in executive control itself, which is why your argument actually is in favour of diagnosis.
Not in the sense that it's definitively tied to a specific pathway disrupted by this or that SNP, but it appears to be heritable in much the same way that height is heritable.
> New high-resolution, three-dimensional maps of the brains of children with attention-deficit/hyperactivity disorder (ADHD) indicate significant and specific anatomical differences within areas of the brain thought to control attentional and inhibitory control systems, compared with brain scans of children without ADHD... The reductions in size of prefrontal regions observed by Sowell and her colleagues are consistent with other reports of reduced frontal lobe volumes in children with ADHD. The more advanced imaging methods and analysis used in the current study, however, suggest that those reductions are localized to more inferior aspects of the prefrontal regions than was previously realized.[1]
> In instances where heredity does not seem to be a factor, difficulties during pregnancy, prenatal exposure to alcohol and tobacco, premature delivery, significantly low birth weight, excessively high body lead levels, and postnatal injury to the prefrontal regions of the brain have all been found to contribute to the risk for ADHD to varying degrees.[2]
> Recently, neuroimaging has led to several important advances in the understanding of the neurobiology underlying the clinical picture of ADHD, and demonstrates a clear brain basis to the disorder in regions involved in attention, and executive and inhibitory control. Furthermore, transcranial magnetic stimulation (TMS) has provided evidence that intracortical inhibition, as indexed by the immature ipsilateral motor cortex, normalises with psychostimulant treatment. There is an exciting confluence between emerging studies in basic neurobiology and the genetic, neuroimaging, and neuropsychological analyses of ADHD.[3]
[1] https://psychnews.psychiatryonline.org/doi/10.1176/pn.39.1.0...
[2] http://www.chadd.org/understanding-adhd/about-adhd/the-scien...